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Systemic application of 3-methyladenine markedly inhibited atherosclerotic lesion in ApoE / mice by modulating autophagy, foam cell formation and immune-negative molecules

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that brand was discontinued in 2008

after its in vivo conversion in arachidonic acid antagonist, and in presence of GSH as biological catalyst, CLA inhibits the formation of Prostaglandin E2, anti-inflammatory mediator